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max in WT synaptoneurosomes, suggesting that Src signaling may very well be downregulated in KI synapses. Then again, our capability to rescue SERT perform in KI midbrain synaptoneurosomes from the inhibition of FAK indicates elevated FAK signaling downstream of the Pro32Pro33 mutant, as verified by amplified pFAK localization in 5-HT synapses. Our